Specialised Acid Wash

SECTION 2: Hazards identification

Hazard statements

This product is classified as: T, Toxic. C, Corrosive. Hazardous according to the criteria of SWA. Dangerous according to the Australian Dangerous Goods (ADG) Code. R20, R33, R35, R37, R24/25. Harmful by inhalation. Danger of cumulative effects. Causes severe burns. Irritating to respiratory system. Toxic in contact with skin and if swallowed

Precautionary statements

S20, S23, S26, $28, S36, S38, $46, $24/25. When using, do not eat or drink. Do not breathe vapours or mists. In case of contact with eyes, rinse immediately with plenty of water and contact a doctor or Poisons Information Centre. After contact with skin, wash immediately with plenty of soap and water, then liberally apply calcium gluconate gel. Wear suitable protective clothing. In case of insufficient ventilation, wear suitable respiratory equipment. If swallowed, contact a doctor or Poisons Information Centre immediately and show this MSDS or label. Avoid contact with skin and eyes.

SUSDP Classification

S7

ADG classification

Class 8 Corrosive Substances. Sub Risk Class 6.1, Toxic Substances

UN Number

2922, CORROSIVE LIQUID, TOXIC, N.O.S Emergency Overview Physical Description & colour: Clear liquid. Odour: Acidic odour.

Health hazards

causes severe burns, toxic in contact with skin and if swallowed, danger of cumulative eftects, harmtul It Inhaled, respiratory tract irrtant. Note: in this context, Hydrotiuoric acid Includes ammonium bifluoride. Hydrofluoric acid burns are a unique clinical entity. Dilute solutions deeply penetrate before dissociating, thus causing delayed injury and symptoms. Burns to the fingers and nail beds may leave the overlying nails intact. Severe burns occur after exposure of concentrated (ie, 50% or stronger solution) Hydrofluoric acid to 1% or more body surface area (BSA), exposure to Hydroflucric acid of any concentration to 5% or more BSA, or inhalation of Hydrofluoric acid fumes from a 60% or stronger solution. The vast majority of cases involve only small areas of exposure, usually on the digits. Solutions of less than 7% may take several hours before onset of symptoms, resulting in delayed presentation, deeper penetration of the undissociated HF acid, and a more severe burn. Pathophysiology: The 2 mechanisms that cause tissue damage are corrosive burn from the free hydrogen ions and chemical burn from tissue penetration of the fluoride ions. Fluoride ions penetrate and form insoluble salts with calcium and magnesium. Soluble salts also are formed with other cations but dissociate rapidly. Consequently, flucride ions release, and further tissue destruction occurs. Mortality/Morbidity: Local effects include tissue destruction and necrosis. Burns may involve underlying bone. Systemic fluoride ion poisoning from severe burns is associated with hypocalcaemia, hyperkalemia, hypomagnesaemia, and sudden death Deaths have been reported from concentrated acid burns to as little as 2.5% BSA. SWA has a publication available, and it can be found at http:/Avww.nohsc.gov.awPDF/Standards/HydrogenFluoride. paf

Potential health effects

Inhalation: Short term exposure: Significant inhalation exposure is considered to be unlikely. product is corrosive to the respiratory tract. Symptoms will include extreme pain in nose and throat and copious secretion of mucous in the nose and throat. Other symptoms such as pulmonary oedema may also become evident, and may be life threatening if exposure is other than brief. Long Term exposure: No data for health effects associated with long term inhalation. Skin Contact: Short term exposure: Concentrated hydrofluoric acid solutions cause immediate pain and produce surface burns similar to those produced by other common acids (eg, erythema, blistering & necrosis). Pain typically is described as deep, burning, or throbbing and often is disproportionate to apparent skin involvement. Solutions of less than 7% may take several hours before onset of symptoms, resulting in delayed presentation, deeper penetration of the undissociated HF acid, and a more severe burn. Long Term exposure: No data for health effects associated with long term skin exposure. Eye Contact: Short term exposure: This product is likely to be mechanically irritating. If exposure is minor or brief, no long term effects should result. However, if material is not removed promptly, scratches to surface of the eye may result with long term consequences. Long Term exposure: No data for health effects associated with long term eye exposure Ingestion: Short term exposure: Mild poisoning causes nausea, vomiting, diarrhoea and abdominal pain. Blood may be vomited. Severe poisoning causes shock, blurred vision, muscle spasm, shallow breathing and convulsions. Kidney failure may occur later. Long Term exposure: Intake of more than 6 mg of flucride per day may result in fluorosis, bone and joint damage. Hypocalcaemia and hypomagnesaemia can occur from absorption of fluoride ion into blood stream. Carcinogen Status: SWA: No significant ingredient is classified as carcinogenic by SWA. NTP: No significant ingredient is classified as carcinogenic by NTP. IARC: Hydrochloric Acid is Class 3 - unclassifiable as to carcinogenicity to humans. See the IARC website for further details. A web address has not been provided as addresses frequently change.