Chromatic �� Primers and Blockouts - Solvent Based

SECTION 2: Hazards identification

OSHA HMIS HEALTH 2 * FLAMMABILITY 3 REACTIVITY 0 0 = Least 1 = Slight 2 = Moderate 3 = High 4 = Extreme

Potential chronic health

Routes of Entry: Inhalation, Ingestion, Skin contact, Eye contact, Absorption.

Medical Conditions Generally Aggravated by Exposure

Eye disease, Skin disease including eczema and sensitization, Kidney disease, Lung disease, Digestive tract disease, Liver disease. ADDITIONAL INGREDIENTS OF PRIMERS & BLOCK OUTS - SOLVENT BASED -- Weight % LIGHT SOLVENT PRODUCT# DENSITY VOC ‡ CALCIUM CRYSTALLINE ETHYL- KAOLIN ALIPHATIC NAPHTHA TALC TITANIUM XYLENE LBS/GL LBS/GL CARBONATE SILICA BENZENE SOLVENT ALIPHATIC, DIOXIDE NAPHTHA MEDIUM 4420101 12.6 2.9 <35 < 1 <10 <15 <10 < 5 <20 4420602 13.0 2.7 <35 <10 <15 <10 < 5 <20 4403101 9.6 3.1 < 1 <15 < 5 Carcinogenicity: IARC No Yes No Yes No NTP No Yes No OSHA No ‡ The VOC content is determined by using a percent solids basis, less water and exempt solvents, for adhesives, coatings and inks and the calculations of EPA Reference Method 24 or equivalent ASTM method approved by the executive office. Immediate (Acute) Health Effects: Inhalation: Can cause moderate respiratory irritation, dizziness, weakness, fatigue, nausea and headache. Can cause severe central nervous system depression (including unconsciousness). Skin Contact: Can cause moderate skin irritation, defatting, and dermatitis. Not likely to cause permanent damage. Eye Contact: Can cause moderate irritation, tearing and reddening, but not likely to permanently injure eye tissue. Skin Absorption: May cause irritation and minor systemic damage. Ingestion: Harmful if swallowed. May cause systemic poisoning. Can cause abdominal discomfort, nausea, vomiting and diarrhea. Aspiration of material into the lungs can cause chemical pneumonitis. Target Organ Acute Toxicity: Respiratory System, Eyes, Skin, Kidneys, Nervous System, Cardiovascular System, Digestive Tract, Stomach, Blood, Liver, Bone Marrow, Lymphatic System, Thyroid, Pituitary, Testes. Long-Term (Chronic) Health Effects: Inhalation: Upon prolonged and/or repeated exposure, can cause moderate respiratory irritation, dizziness, weakness, fatigue, nausea and headache. Skin Contact: Upon prolonged or repeated contact, can cause moderate skin irritation, defatting, and dermatitis. Not likely to cause permanent damage. Eye Contact: Upon prolonged or repeated contact, can cause moderate irritation, tearing and reddening, but not likely to permanently injure eye tissue. Skin Absorption: Upon prolonged or repeated exposure, harmful if absorbed through the skin. May cause severe irritation and systemic damage. Carcinogenicity: Target Organ Chronic Toxicity: Respiratory System, Nervous System, Kidneys, Eyes, Skin, Cardiovascular System, Blood, Liver, Digestive Tract, Lymphatic System, Pituitary, Testes. NOTICE - Reports have associated repeated and prolonged occupational overexposure to solvents with brain and nervous system damage. Intentional misuse by deliberately concentrating and inhaling the contents may be harmful or fatal. Lifetime inhalation exposure of rats and mice to high concentrations of ethylbenzene (750 ppm) resulted in increases in certain types of cancer, including kidney, lung and liver tumors. Testicular adenomas were increased as were thyroid effects in rats at 750 ppm. Pituitary effects were observed in female mice at 250 ppm. These effects were absent when exposure was below 75 ppm ethylbenzene. The study does not address the relevance of these results to humans. or grinding dried or cured material may release particles of crystalline silica (quartz). Exposure to airborne particles may cause lung damage including a risk of cancer. Chronic exposure may result in chest pain, difficulty breathing, lung damage and silicosis. (Silicosis is the permanent deposition of silica in lung tissue that results in lung damage.) There may exist a relationship between silicosis and certain cancers. IARC has recently re-evaluated titanium dioxide as possibly carcinogenic to humans (Group 2B) based on animal studies. However, human studies available to date do not suggest that occupational exposure to titanium dioxide increases cancer risk. The ACGIH classifies titanium dioxide as A4 (not classifiable as a human carcinogen). NTP does not classify it as carcinogenic. IARC's evaluation shows inadequate evidence of carcinogenicity in humans, but sufficient evidence of carcinogenicity in experimental animals. The evidence shows that high concentrations of powdered and ultrafine titanium dioxide dust caused respiratory tract cancer in rats exposed by either natural inhalation or direct introduction into the lungs. However, the same results are observed in people working in dusty environments. Therefore, IARC extended this idea to workers with exposures to titanium dioxide dust, if there are insufficient dust control measures in place. Based on the IARC decision, Canadian officials have agreed that titanium dioxide is classifiable as WHMIS D2A (carcinogen), and that it is not necessary to wait for release of the full monograph. OSHA requires the status on US MSDSs to change within 90 days of publication in the IARC monograph volume 93. This product contains pigments which may become a dust nuisance when removed by abrasive blasting, sanding or grinding. IV. FIRST AID Inhalation: Remove to fresh air. If breathing is difficult, have a trained individual administer oxygen. Seek medical attention if symptoms persist.