Chromatic �� Primers and Block Outs - Water Based

SECTION 2: Hazards identification

OSHA HMIS HEALTH 1 * FLAMMABILITY 1 REACTIVITY 0 0 = Least 1 = Slight 2 = Moderate 3 = High 4 = Extreme

Potential chronic health

Routes of Entry: Inhalation, Ingestion, Skin contact, Eye contact, Absorption.

Medical Conditions Generally Aggravated by Exposure

Lung disease, Eye disease, Skin disease including eczema and sensitization, Digestive tract disease, Liver disease. ADDITIONAL INGREDIENTS OF PRIMERS & BLOCK OUTS - WATER BASED -- Weight % ETHYLENE GLYCOL 1-METHOXY- DENSITY V.O.C. ‡ CRYSTALLINE DIBUTYL MONO-n- CYCLIC FUMED PROPYLENE TITANIUM 2-HYDROXY- PRODUCT# LBS/GL SILICA PHTHALATE BUTYL ETHER MICA TALC AMINE SILICA GLYCOL DIOXIDE PROPANE 4411002 11.9 1 < 1 <10 <15 < 5 <25 4411008 10.3 2.7 < 5 <15 < 5 <25 4411010 10.7 < 5 <10 < 5 <25 4331010 8.6 2.4 < 5 <10 < 5 4331011 8.6 2.4 < 5 <10 < 5 Carcinogenicity: IARC No Yes No NTP Yes No OSHA No ‡ The VOC content is determined by using a percent solids basis, less water and exempt solvents, for adhesives, coatings and inks and the calculations of EPA Reference Method 24 or equivalent ASTM method approved by the executive office. Immediate (Acute) Health Effects: Inhalation: Can cause moderate respiratory irritation, dizziness, weakness, fatigue, nausea and headache. Harmful. Can cause systemic damage, see target organs below. When Zinc Oxide is present, this product may cause metal fume fever with resulting flu-like symptoms. Can cause systemic damage, see target organs below. Skin Contact: Can cause moderate skin irritation, defatting, and dermatitis. Not likely to cause permanent damage. Eye Contact: Contact with the eyes may cause moderate to severe eye injury. Eye contact may result in tearing and reddening, but not likely to permanently injure eye tissue. Temporary vision impairment (cloudy or blurred vision) is possible. Skin Absorption: Harmful if absorbed through the skin. May cause severe irritation and systemic damage. Ingestion: Toxic if swallowed. May cause target organ failure and/or death. Can cause abdominal discomfort, nausea, vomiting and diarrhea. Ingestion of this product may result in central nervous system effects including headache, sleepiness, dizziness, slurred speech and blurred vision. Target Organ Acute Toxicity: Respiratory System, Eyes, Cardiovascular System, Skin, Digestive Tract, Liver, Blood, Kidneys, Lymphoid System. Long-Term (Chronic) Health Effects: Inhalation: Upon prolonged and/or repeated exposure, can cause moderate respiratory irritation, dizziness, weakness, fatigue, nausea and headache. Skin Contact: Upon prolonged or repeated contact, can cause moderate skin irritation, defatting, and dermatitis. Not likely to cause permanent damage. Eye Contact: Upon prolonged or repeated contact, can cause moderate to severe eye injury. Eye contact may result in tearing and reddening, but not likely to permanently injure eye tissue. Temporary vision impairment (cloudy or blurred vision) is possible. Skin Absorption: Upon prolonged or repeated exposure, harmful if absorbed through the skin. May cause severe irritation and systemic damage. Carcinogenicity: Target Organ Chronic Toxicity: Respiratory System, Eyes, Cardiovascular System, Kidneys, Nervous System, Skin, Digestive Tract, Liver, Blood. NOTICE - Reports have associated repeated and prolonged occupational overexposure to solvents with brain and nervous system damage. Intentional misuse by deliberately concentrating and inhaling the contents may be harmful or fatal. Over exposure of laboratory animals to a high concentration (700 ppm for 7 hours) of ethylene glycol n-butyl ether caused systemic toxicity in the form of hemoglobinuria and lung, kidney and liver changes. Exposure of rats to a lower concentration (320 ppm) for five weeks caused hemolytic anemia and increased fragiliity of the red blood cells. However, dogs exposed to a higher concentration (400 ppm) for a longer period (12 weeks) showed only slight injury. Humans appear to be less susceptible, and toxicity may be more likely to occur as a result of skin absorption than from inhalation. IARC has recently re-evaluated titanium dioxide as possibly carcinogenic to humans (Group 2B) based on animal studies. However, human studies available to date do not suggest that occupational exposure to titanium dioxide increases cancer risk. The ACGIH classifies titanium dioxide as A4 (not classifiable as a human carcinogen). NTP does not classify it as carcinogenic. IARC's evaluation shows inadequate evidence of carcinogenicity in humans, but sufficient evidence of carcinogenicity in experimental animals. The evidence shows that high concentrations of powdered and ultrafine titanium dioxide dust caused respiratory tract cancer in rats exposed by either natural inhalation or direct introduction into the lungs. However, the same results are observed in people working in dusty environments. Therefore, IARC extended this idea to workers with exposures to titanium dioxide dust, if there are insufficient dust control measures in place. Based on the IARC decision, Canadian officials have agreed that titanium dioxide is classifiable as WHMIS D2A (carcinogen), and that it is not necessary to wait for release of the full monograph. OSHA requires the status on US MSDSs to change within 90 days of publication in the IARC monograph volume 93. This product contains pigments which may become a dust nuisance when removed by abrasive blasting, sanding or grinding. IV. FIRST AID Inhalation: Remove to fresh air. If breathing is difficult, have a trained individual administer oxygen. Seek medical attention if symptoms persist.